GIPR/CRE-Luc/HEK293-南京科佰生物科技有限公司

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联系人：蒋经理
电话：86-25-86880024
号码：86-25-86880024
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地址：南京市雨花区长虹路222号
Email： duhaitao@cobioer.com

CBP71346
I. Background
The gastric inhibitory polypeptide receptor (GIP-R), also known as the glucose-dependent insulinotropic polypeptide receptor,which was originally identified as an activity in gut extracts that inhibited gastric acid secretion and gastrin release, but subsequently was demonstrated to stimulate insulin release in the presence of elevated glucose. Mice lacking this gene exhibit higher blood glucose levels with impaired initial insulin response after oral glucose load. Defect in this gene thus may contribute to the pathogenesis of diabetes.

II. Introduction
Host Cell:	HEK293
Stability:	20 passages (in-house test, that not means the cell line will be instable beyond the passages we tested.)
Freeze Medium:	90% FBS+10% DMSO
Culture Medium:	DMEM +10%FBS+2ug/ml puromycin+200ug/ml hygromycin
Mycoplasma Status:	Negative
Storage:	Liquid nitrogen immediately upon delivery
Transducer:	Gs

Ⅲ. Description of Host Cell Line
Organism:	Human
Tissue:	Embryonic kidney
Morphology:	Epithelial
Growth Properties:	Adherent

Ⅳ. Representative Data
Figure 1. Recombinant GIPR/CRE-Luc/HEK293 stably expressing human GIPR. Figure 2. GIPR/CRE Reporter - HEK293 Recombinant Cell Line (C37).